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Literature summary for 1.1.1.284 extracted from

  • Wen, D.; Sun, S.; Yang, W.; Zhang, L.; Liu, S.; Gong, B.; Shi, Q.
    Overexpression of S-nitrosoglutathione reductase alleviated iron-deficiency stress by regulating iron distribution and redox homeostasis (2019), J. Plant Physiol., 237, 1-11 .
    View publication on PubMed
Search for more literature for 1.1.1.284

Cloned(Commentary)

Cloned (Comment) Organism
gene ADH3, recombinant overexpression of enzyme GSNOR in Solanum lycopersicum using Agrobacterium-mediated transformation of the CaMV35S::GSNOR overexpression construct, quantitative real-time PCR enzyme expression analysis Solanum lycopersicum

Protein Variants

Protein Variants Comment Organism
additional information there are no significant changes in the physiological or chlorophyll fluorescence parameters between tomato leaves of wild-type and transgenic GSNOR overexpressing lines under control conditions. In contrast, apparent chlorosis is observed in the wild-type lines after 20 d under Fe-deficiency conditions, while transgenic lines show significantly higher Fe-deficiency tolerance levels than wild-type lines as assessed by morphological characteristics, especially the color of the stem apices and new leaves Solanum lycopersicum

Localization

Localization Comment Organism GeneOntology No. Textmining
chloroplast
-
 Solanum lycopersicum 9507
-

Metals/Ions

Metals/Ions Comment Organism Structure
Fe2+ GSNOR expression is induced by Fe deficiency in tomato leaves and roots, while its overexpression alleviates chlorosis under Fe-deficiency conditions. GSNOR overexpression positively regulates the Fe distribution from root to shoot, which might result from the transcriptional regulation of genes involved in Fe metabolism Solanum lycopersicum
Zn2+ required Solanum lycopersicum

Organism

Organism UniProt Comment Textmining
Solanum lycopersicum D2Y3F4
-
-

Source Tissue

Source Tissue Comment Organism Textmining
leaf
-
 Solanum lycopersicum
-
root
-
 Solanum lycopersicum
-
shoot
-
 Solanum lycopersicum
-

Synonyms

Synonyms Comment Organism
GSNOR
-
 Solanum lycopersicum
S-nitrosoglutathione reductase
-
 Solanum lycopersicum

Temperature Optimum [°C]

Temperature Optimum [°C] Temperature Optimum Maximum [°C] Comment Organism
25
-
 assay at Solanum lycopersicum

pH Optimum

pH Optimum Minimum pH Optimum Maximum Comment Organism
7.2
-
 assay at Solanum lycopersicum

Expression

Organism Comment Expression
Solanum lycopersicum GSNOR expression is induced by Fe deficiency in tomato up

General Information

General Information Comment Organism
malfunction overexpression alleviates chlorosis under Fe-deficiency conditions. GSNOR overexpression positively regulates the Fe distribution from root to shoot, which might result from the transcriptional regulation of genes involved in Fe metabolism. Overexpression of GSNOR maintains redox homeostasis and protects chloroplasts from Fe deficiency-related damage, resulting in a greater photosynthetic capacity. As a nitric oxide regulator, GSNOR's overexpression decreases the excessive accumulation of nitric oxide and S-nitrosothiols during the Fe deficiency, and maintains the homeostases of reactive oxygen species and reactive nitrogen species. Moreover, GSNOR overexpression, probably at the level of genes and proteins, along with protein S-nitrosylation, promotes Fe uptake and regulates the shoot/root Fe ratio under Fe-deficiency conditions. The overexpression of GSNOR alleviates the Fe-deficiency-induced oxidative stress Solanum lycopersicum
physiological function enzyme GSNOR is a nitric oxide regulator and is involved in responses to iron deficiency, GSNOR-regulated RNS homeostasis under Fe-deficiency conditions, overview. Ferric-chelate reductase activity is regulated by GSNOR Solanum lycopersicum